mutant human 2b-ar lacking 16 amino acids (Liggett Group Inc)
90
Structured Review
Liggett Group Inc
mutant human 2b-ar lacking 16 amino acids
Mutant Human 2b Ar Lacking 16 Amino Acids, supplied by Liggett Group Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mutant+human+2b-ar+lacking+16+amino+acids/mutant+human+2b+ar+lacking+16+amino+acids/pm16533872-252-6-28
Average 90 stars, based on 1 article reviews
Mutant Human 2b Ar Lacking 16 Amino Acids, supplied by Liggett Group Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mutant+human+2b-ar+lacking+16+amino+acids/mutant+human+2b+ar+lacking+16+amino+acids/pm16533872-252-6-28
Average 90 stars, based on 1 article reviews
mutant human 2b-ar lacking 16 amino acids - by Bioz Stars,
2026-09
90/100 stars
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Related Articles
Mutagenesis:Article Title: Involvement of G protein-coupled receptor kinase (GRK) 3 and GRK2 in down-regulation of the alpha2B-adrenoceptor. Article Snippet: Increasing the cellular levels of G protein-coupled receptor kinase (GRK) 2 or GRK3 renders the 2B-adrenoceptor (AR) more sensitive to agonist-induced down-regulation (J Pharmacol Exp Ther 312:767–773, 2005).. However, an absolute requirement of GRK3 and GRK2 for 2B-AR down-regulation is controversial.. In this study, using NG108 cells (endogenous 2B-AR), we provide strong evidence for a critical role of both GRK3 and GRK2 in down-regulation of the 2B-AR. Phospho-proteomics:Article Title: Involvement of G protein-coupled receptor kinase (GRK) 3 and GRK2 in down-regulation of the alpha2B-adrenoceptor. Article Snippet: Increasing the cellular levels of G protein-coupled receptor kinase (GRK) 2 or GRK3 renders the 2B-adrenoceptor (AR) more sensitive to agonist-induced down-regulation (J Pharmacol Exp Ther 312:767–773, 2005).. However, an absolute requirement of GRK3 and GRK2 for 2B-AR down-regulation is controversial.. In this study, using NG108 cells (endogenous 2B-AR), we provide strong evidence for a critical role of both GRK3 and GRK2 in down-regulation of the 2B-AR. |